Dr. David Timothy Berg D.O.
Neurologist | Neuromusculoskeletal Medicine, Sports Medicine
3600 30th St Des Moines IA, 50310About
Dr. David Berg is a distinguished Neurologist in Des Moines, IA. Dr. Berg specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Berg employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Berg is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Des Moines University College of Osteopathic Medicine and Health Sciences 1981
Des Moines University College of Osteopathic Medicine 1981
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Colorectal cancer in a 43-year-old man.
- 17beta-estradiol, but not raloxifene, decreases thrombomodulin in the antithrombotic protein C pathway.
- Therapeutic options for treating advanced colorectal cancer.
- Differential mechanisms of plasminogen activator inhibitor-1 gene activation by transforming growth factor-beta and tumor necrosis factor-alpha in endothelial cells.
- Modulation of thrombomodulin-dependent activation of human protein C through differential expression of endothelial Smads.
- Engineering the proteolytic specificity of activated protein C improves its pharmacological properties.
- 5' sequence of vesicular stomatitis virus N-gene confers selective translation of mRNA.
- Evaluation of protein C and other biomarkers as predictors of mortality in a rat cecal ligation and puncture model of sepsis.
- Smad6s regulates plasminogen activator inhibitor-1 through a protein kinase C-beta-dependent up-regulation of transforming growth factor-beta.
- Intensive post-remission therapy with Ara-C in adults with acute myeloid leukemia: initial results of a CALGB phase III trial. The Cancer and Leukemia Group B.
- FoxA2 involvement in suppression of protein C, an outcome predictor in experimental sepsis.
- Role of protein C in renal dysfunction after polymicrobial sepsis.
- Activated protein C suppresses adrenomedullin and ameliorates lipopolysaccharide-induced hypotension.
- Negative regulation of inducible nitric-oxide synthase expression mediated through transforming growth factor-beta-dependent modulation of transcription factor TCF11.
- Activated protein C decreases tumor necrosis factor related apoptosis-inducing ligand by an EPCR- independent mechanism involving Egr-1/Erk-1/2 activation.
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