Robert James Bache M.D.
Cardiologist | Cardiovascular Disease
University Of Minnesota Physicians 516 Delaware Street Minneapolis MN, 55455About
Dr. Robert Bache is a cardiologist practicing in Minneapolis, MN. Dr. Bache specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Bache also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Harvard Med Sch, Boston Ma 1964
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Regulation of myocardial blood flow by oxygen consumption is maintained in the failing heart during exercise.
- Myocardial creatine kinase kinetics in hearts with postinfarction left ventricular remodeling.
- Effect of NO on transmural distribution of blood flow in hypertrophied left ventricle during exercise.
- Role of K(+)(ATP) channels and adenosine in regulation of coronary blood flow in the hypertrophied left ventricle.
- Nitroglycerin dilates coronary collateral vessels during exercise after blockade of endogenous NO production.
- Regulation of coronary vasomotor tone under normal conditions and during acute myocardial hypoperfusion.
- Coronary nitric oxide production in response to exercise and endothelium-dependent agonists.
- Intracoronary collagen: effects on coronary collateral circulation and the role of thromboxane.
- Effect of sildenafil on coronary active and reactive hyperemia.
- Regional myocardial blood flow during graded treadmill exercise in the dog.
- Cyclic nucleotide phosphodiesterase type 5 activity limits blood flow to hypoperfused myocardium during exercise.
- Myocardial oxygenation and high-energy phosphate levels during graded coronary hypoperfusion.
- Noninvasive measurements of transmural myocardial metabolites using 3-D (31)P NMR spectroscopy.
- Myocardial creatine kinase kinetics and isoform expression in hearts with severe LV hypertrophy.
- Selective blockade of mitochondrial K(ATP) channels does not impair myocardial oxygen consumption.
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