Dr. Dr. Ed Warren Childs, MD
Surgeon
75 Piedmont Ave Ne Suite 700 Atlanta GA, 30303About
Dr. Ed Childs is a general surgeon practicing in Atlanta, GA. Dr. Childs specializes in abdominal contents including the esophagus, stomach, liver, gallbladder, pancreas and often thyroid glands. General surgeons are able to deal with almost any surgical or critical care emergency, also involving the skin or soft tissue trauma. Dr. Childs provides quality surgical service for gravely ill or injured patients and is able to respond quickly due to knowledge of various surgical procedures.
Education and Training
Tx A & M Univ Coll Of Med- College Station Tx 1989
Texas A&M University / Main Campus 1989
Board Certification
SurgeryAmerican Board of SurgeryABS
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Adrenal crisis after traumatic bilateral adrenal hemorrhage.
- Traumatic lumbar artery pseudoaneurysm presenting with massive lower gastrointestinal tract bleeding.
- Role of herniography in the diagnosis of occult hernias.
- Morphine sulfate attenuates hemorrhagic shock-induced hyperpermeability.
- Apoptotic signaling induces hyperpermeability following hemorrhagic shock.
- The influence of early hemodynamic optimization on biomarker patterns of severe sepsis and septic shock.
- Alpha lipoic acid attenuates microvascular endothelial cell hyperpermeability by inhibiting the intrinsic apoptotic signaling.
- Morphine attenuates microvascular hyperpermeability via a protein kinase
- PKC and MLCK-dependent, cytokine-induced rat coronary endothelial dysfunction.
- Alpha-lipoic acid attenuates hemorrhagic shock-induced apoptotic signaling and vascular hyperpermeability.
- 17beta-estradiol mediates protection against microvascular endothelial cell hyperpermeability.
- Tonsillar abscess.
- Cyclosporine A prevents vascular hyperpermeability after hemorrhagic shock by inhibiting apoptotic signaling.
- (-)-Deprenyl inhibits vascular hyperpermeability after hemorrhagic shock.
- (-)-Deprenyl inhibits thermal injury-induced apoptotic signaling and hyperpermeability in microvascular endothelial cells.
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