Dr. Michael N Sack M.D.
Doctor
Nhlbi Nih Bld 10-Crc, Rm 53150 Bethesda MD, 20892About
Dr. Michael N Sack M.D. is a top Doctor in Bethesda, MD. With a passion for the field and an unwavering commitment to their specialty, Dr. Michael N Sack M.D. is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Dr. Michael N Sack M.D. is a prime example of a true leader in health care. As a leader and expert in their field, Dr. Michael N Sack M.D. is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In Bethesda, MD, Dr. Michael N Sack M.D. is a true asset to their field and dedicated to the profession of medicine.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Tumor necrosis factor in myocardial hypertrophy and ischaemia--an anti-apoptotic
- Coordinate regulation of metabolic enzyme encoding genes during cardiac development and following carvedilol therapy in spontaneously hypertensive rats.
- Insulin administered at reoxygenation exerts a cardioprotective effect in myocytes by a possible anti-apoptotic mechanism.
- Dinitrophenol, cyclosporin A, and trimetazidine modulate preconditioning in the isolated rat heart: support for a mitochondrial role in cardioprotection.
- Apoptosis: a pivotal event or an epiphenomenon in the pathophysiology of heart failure?
- Mechanisms whereby glucose deprivation triggers metabolic preconditioning in the isolated rat heart.
- Delta opioid receptor stimulation mimics ischemic preconditioning in human heart muscle.
- Enhanced angiotensin II activity in heart failure: reevaluation of the counterregulatory hypothesis of receptor subtypes.
- TNF alpha is required for hypoxia-mediated right ventricular hypertrophy.
- Ischemic and pharmacological preconditioning in Girardi cells and C2C12 myotubes induce mitochondrial uncoupling.
- Myocardial protection by insulin at reperfusion requires early administration and is mediated via Akt and p70s6 kinase cell-survival signaling.
- Identification of a novel role for sphingolipid signaling in TNF alpha and ischemic preconditioning mediated cardioprotection.
- Innate immunity and cardiac preconditioning: a putative intrinsic cardioprotective program.
- Classic ischemic but not pharmacologic preconditioning is abrogated following genetic ablation of the TNFalpha gene.
- Metabolic plasticity and the promotion of cardiac protection in ischemia and ischemic preconditioning.
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