Mrs. Lisa Dawn Mcdaniel MS,CCC-SLP
Speech-Language Pathologist
5313 IVY RIDGE CT NEWBURGH IN, 47630About
Dr. Lisa Mcdaniel is a speech language pathologist practicing in NEWBURGH, IN. Dr. Mcdaniel specializes in speech, language and swallowing disorders in patients. As a speech language pathologist, Dr. Mcdaniel evaluates, diagnoses and treats patients with communication and swallowing troubles. These conditions may be due to developmental delay, brain injury, hearing loss, autism, stroke or other diseases and injuries. Dr. Mcdaniel helps patients make sounds and improve their voices through various methods. Speech language pathologists also work with patients to strengthen muscles used to speak and swallow, and work with individuals and families to help cope with their conditions.
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Expert Publications
Data provided by the National Library of Medicine- DNA damage responses protect xeroderma pigmentosum variant from UVC-induced clastogenesis.
- Chromosome instability and tumor predisposition inversely correlate with BLM protein levels.
- Mutations in the Trp53 gene of UV-irradiated Xpc mutant mice suggest a novel Xpc-dependent DNA repair process.
- The role of endogenous and exogenous DNA damage and mutagenesis.
- Mapping of a single locus capable of complementing the defective heterochromatin phenotype of Roberts syndrome cells.
- Transcription-associated breaks in xeroderma pigmentosum group D cells from patients with combined features of xeroderma pigmentosum and Cockayne syndrome.
- Telomerase-immortalized human fibroblasts retain UV-induced mutagenesis and p53-mediated DNA damage responses.
- Elevated mutation rates in the germline of Polkappa mutant male mice.
- TERF2-XPF: caught in the middle; beginnings from the end.
- DNA polymerases for translesion DNA synthesis: enzyme purification and mouse models for studying their function.
- A novel XPC pathogenic variant detected in archival material from a patient diagnosed with Xeroderma Pigmentosum: a case report and review of the genetic variants reported in XPC.
- Validation of XP-C pathogenic variations in archival material from a live XP patient.
- XPF/ERCC4 and ERCC1: their products and biological roles.
- Polk mutant mice have a spontaneous mutator phenotype.
- Loss of Blm enhances basal cell carcinoma and rhabdomyosarcoma tumorigenesis in Ptch1+/- mice.
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