Dr. Alice P Pentland MD
Dermatologist
601 Elmwood Ave Box 656 Rochester NY, 14642About
Dr. Alice Pentland is a dermatologist practicing in Rochester, NY. Dr. Pentland specializes in skin care. Dermatologists evaluate and manage both common and uncommon skin conditions. These conditions include acne, psoriasis, warts, skin infections, atopic dermatitis, herpes simplex and more. Dermatologists are also experts in more complex skin diseases like impetigo, hidradenitis and milaria. Dr. Pentland diagnoses skin problems and develops unique treatments plans for each individual patient.
Education and Training
Univ of Mi Med Sch, Ann Arbor Mi 1978
University of Mi Med Sch 1978
University of Michigan Medical School 1978
Board Certification
DermatologyAmerican Board of DermatologyABD
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Immunohistochemical analysis of cyclooxygenase expression in human skin.
- Reduction of UV-induced skin tumors in hairless mice by selective COX-2 inhibition.
- Coordinate expression of secretory phospholipase A(2) and cyclooxygenase-2 in activated human keratinocytes.
- Inventing the future--tools for self health.
- Cyclooxygenase inhibitors for skin cancer prevention: are they beneficial enough?
- Loss of the EP2 prostaglandin E2 receptor in immortalized human keratinocytes results in increased invasiveness and decreased paxillin expression.
- Pertussis toxin-sensitive secretory phospholipase A2 expression and motility in activated primary human keratinocytes.
- Epidermal COX-2 induction following ultraviolet irradiation: suggested mechanism for the role of COX-2 inhibition in photoprotection.
- Cyclooxygenase-1 deletion enhances apoptosis but does not protect against ultraviolet light-induced tumors.
- Immunolocalization of low-affinity prostaglandin E receptors, EP and EP, in adult human epidermis.
- The EP3 receptor stimulates ceramide and diacylglycerol release and inhibits growth of primary keratinocytes.
- Selective cyclooxygenase-2 inhibition produces heterogeneous erythema response to ultraviolet irradiation.
- sPLA2-X stimulates cutaneous melanocyte dendricity and pigmentation through a lysophosphatidylcholine-dependent mechanism.
- Deletion of prostaglandin E2 EP2 receptor protects against ultraviolet-induced carcinogenesis, but increases tumor aggressiveness.
- Sequential down-regulation of E-cadherin with squamous cell carcinoma progression: loss of E-cadherin via a prostaglandin E2-EP2 dependent posttranslational mechanism.
Treatments
- Psoriasis
- Eczema
- Dermatitis
- Moles
- Warts
- Actinic Keratosis
- Birth Defects
- Birthmark
- Contact Dermatitis
- Genital Warts
- Seborrheic Keratosis
- Skin Tag
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