Dr. Jodie Lynn Babitt MD
Nephrologist (Kidney Specialist) | Nephrology
55 Fruit St Grb 1003 Massachusetts Genera Boston MA, 02114About
Dr. Jodie Babitt is a nephrologist practicing in Boston, MA. Dr. Babitt specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Babitt most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Harvard Medical School 1999
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- BMP6 is a key endogenous regulator of hepcidin expression and iron metabolism.
- Bone morphogenetic protein signaling is impaired in an HFE knockout mouse model of hemochromatosis.
- BMP6 treatment compensates for the molecular defect and ameliorates hemochromatosis in Hfe knockout mice.
- Serum and liver iron differently regulate the bone morphogenetic protein 6 (BMP6)-SMAD signaling pathway in mice.
- Repulsive guidance molecule (RGM) family proteins exhibit differential binding kinetics for bone morphogenetic proteins (BMPs).
- A hepcidin lowering agent mobilizes iron for incorporation into red blood cells in an adenine-induced kidney disease model of anemia in rats.
- The iron cycle in chronic kidney disease (CKD): from genetics and experimental models to CKD patients.
- Hemojuvelin and bone morphogenetic protein (BMP) signaling in iron homeostasis.
- MicroRNA-130a is up-regulated in mouse liver by iron deficiency and targets the bone morphogenetic protein (BMP) receptor ALK2 to attenuate BMP signaling and hepcidin transcription.
- Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production.
- Activin B Induces Noncanonical SMAD1/5/8 Signaling via BMP Type I Receptors in Hepatocytes: Evidence for a Role in Hepcidin Induction by Inflammation in Male Mice.
- Hepcidin regulation in the anemia of inflammation.
- The SMAD Pathway Is Required for Hepcidin Response During Endoplasmic Reticulum Stress.
- Ironing out the cross talk between FGF23 and inflammation.
- Endothelial cells produce bone morphogenetic protein 6 required for iron homeostasis in mice.
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