Dr. Richard Ian Enelow MD
Pulmonologist | Pulmonary Disease
1 Medical Center Dr Lebanon NH, 03756About
Dr. Richard Enelow practices Pulmonology in Lebanon, NH. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Enelow manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Boston University School of Medicine 1983
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Pulmonary Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Perforin-independent CD8(+) T-cell-mediated cytotoxicity of alveolar epithelial cells is preferentially mediated by tumor necrosis factor-alpha: relative insensitivity to Fas ligand.
- Hemorrhage and resuscitation induce delayed inflammation and pulmonary dysfunction in mice.
- Alveolar epithelial cell chemokine expression triggered by antigen-specific cytolytic CD8(+) T cell recognition.
- Alveolar epithelial cell chemokine expression induced by specific antiviral CD8+ T-cell recognition plays a critical role in the perpetuation of experimental interstitial pneumonia.
- Type II pneumocyte-CD8+ T-cell interactions. Relationship between target cell cytotoxicity and activation.
- CD8(+) T cell-mediated injury in vivo progresses in the absence of effector T cells.
- Cytokine-induced human multinucleated giant cells have enhanced candidacidal
- Cutting edge: pulmonary immunopathology mediated by antigen-specific expression of TNF-alpha by antiviral CD8+ T cells.
- CD4 T Lymphocyte-mediated lung disease: steady state between pathological and tolerogenic immune reactions.
- Regional and generalized changes in cytosolic free calcium in monocytes during phagocytosis.
- Requirement for tumor necrosis factor-receptor 2 in alveolar chemokine expression depends upon the form of the ligand.
- Regulation of gene expression in RAW 264.7 macrophage cell line by interferon-gamma.
- Lung epithelial NF-kappaB and Stat1 signaling in response to CD8+ T cell antigen recognition.
- Cellular immunity and lung injury in respiratory virus infection.
- Induction of multinucleated giant cell formation from in vitro culture of human monocytes with interleukin-3 and interferon-gamma: comparison with other stimulating factors.
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