Dr. David Gershon Cook M.D.
Neurologist | Neurology
105 Llanfair Rd Ardmore PA, 19003Rating
5/5
About
Dr. David Cook is a distinguished Neurologist in Ardmore, PA. Dr. Cook specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Cook employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Cook is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Perelman School of Medicine University of Pennsylvania 1968
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Endoproteolysis of beta-secretase (beta-site amyloid precursor protein-cleaving enzyme) within its catalytic domain. A potential mechanism for regulation.
- Presenilin-1 and intracellular calcium stores regulate neuronal glutamate uptake.
- Presenilin 1 deficiency alters the activity of voltage-gated Ca2+ channels in cultured cortical neurons.
- Detergent-insoluble EAAC1/EAAT3 aberrantly accumulates in hippocampal neurons of Alzheimer's disease patients.
- Peripheral amyloid-beta levels regulate amyloid-beta clearance from the central nervous system.
- Neuronal expression of splice variants of "glial" glutamate transporters in brains afflicted by Alzheimer's disease: unmasking an intrinsic neuronal property.
- Cerebrocerebellar hypometabolism associated with repetitive blast exposure mild traumatic brain injury in 12 Iraq war Veterans with persistent post-concussive symptoms.
- Aberrant detergent-insoluble excitatory amino acid transporter 2 accumulates in
- Chronic dysfunction of astrocytic inwardly rectifying K+ channels specific to the neocortical epileptic focus after fluid percussion injury in the rat.
- GLT-1 loss accelerates cognitive deficit onset in an Alzheimer's disease animal model.
- Localisation of novel forms of glutamate transporters and the cystine-glutamate antiporter in the choroid plexus: Implications for CSF glutamate homeostasis.
- Differential expression of the glutamate transporter GLT-1 in pancreas.
- Amyloid-β1-42 slows clearance of synaptically released glutamate by mislocalizing astrocytic GLT-1.
- Blast exposure causes early and persistent aberrant phospho- and cleaved-tau expression in a murine model of mild blast-induced traumatic brain injury.
- Partial Loss of the Glutamate Transporter GLT-1 Alters Brain Akt and Insulin Signaling in a Mouse Model of Alzheimer's Disease.
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