George A Stouffer MD
Cardiologist | Cardiovascular Disease
101 MANNING DR CHAPEL HILL ND, 27599About
Dr. George Stouffer is a cardiologist practicing in CHAPEL HILL, ND. Dr. Stouffer specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Stouffer also practices preventative medicine, helping patients maintain a heart-healthy life.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- New treatment strategies in patients with impaired left ventricular systolic function. Part I: Background and treatment of mild cardiac dysfunction.
- New treatment strategies in patients with impaired left ventricular systolic function. Part II: Treatment of moderate to severe cardiac dysfunction.
- Interesting cases from the University of Texas Medical Branch.
- Use of glycoprotein IIb/IIIa inhibitors in patients with coronary artery disease.
- Use of coronary flow reserve to evaluate the physiologic significance of coronary artery disease.
- Left main coronary artery disease.
- Use of intraluminal stents in the treatment of carotid, renal, and peripheral arterial disease.
- Protein complexes involving alpha v beta 3 integrins, nonmuscle myosin heavy chain-A, and focal adhesion kinase from in thrombospondin-treated smooth muscle cells.
- Left main coronary artery disease in adults younger than 50 years: a comparison with older patients.
- Autocrine thrombospondin partially mediates TGF-beta1- induced proliferation of vascular smooth muscle cells.
- A prospective evaluation of angiography-guided coronary stent implantation with high versus very high balloon inflation pressure.
- Morphological patterns of death by myocytes in arrhythmogenic right ventricular dysplasia.
- Focal elastic obstruction of the inferior vena cava.
- Mitral valve prolapse: a review of the literature.
- Eptifibatide and 7E3, but not tirofiban, inhibit alpha(v)beta(3) integrin-mediated binding of smooth muscle cells to thrombospondin and prothrombin.
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