Mary Elizabeth Kyle DPT
Physical Therapist
25250 75th St Salem WI, 53168About
Mary Kyle is a physical therapist practicing in Salem, WI. Mary Kyle specializes in physical treatment to help a patient reduce pain, restore mobility, rehabilitate an injury, or increase movement and overall function. As a physical therapist, Mary Kyle can treat multiple conditions with exercises, ultrasound, electrical stimulation, joint mobilization, heat, ice, massage, laser or light therapy and more. Mary Kyle will create a treatment plan based on the patients specific injury or condition, and might target a specific body part or body system based on the individual.
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Expert Publications
Data provided by the National Library of Medicine- The killing of cultured hepatocytes by N-acetyl-p-benzoquinone imine (NAPQI) as a model of the cytotoxicity of acetaminophen.
- Mechanisms of cell injury by activated oxygen species.
- L-carnitine delays the killing of cultured hepatocytes by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine.
- Autophagic degradation of protein generates a pool of ferric iron required for the killing of cultured hepatocytes by an oxidative stress.
- Metabolism of acetaminophen by cultured rat hepatocytes. Depletion of protein thiol groups without any loss of viability.
- Protein thiol depletion and the killing of cultured hepatocytes by hydrogen peroxide.
- The inhibition of lipid peroxidation by disulfiram prevents the killing of cultured hepatocytes by allyl alcohol, tert-butyl hydroperoxide, hydrogen peroxide and diethyl maleate.
- Mitochondrial damage as a mechanism of cell injury in the killing of cultured hepatocytes by tert-butyl hydroperoxide.
- tert-butyl hydroperoxide kills cultured hepatocytes by peroxidizing membrane lipids.
- Superoxide dismutase and catalase protect cultured hepatocytes from the cytotoxicity of acetaminophen.
- 1,3-(2-Chloroethyl)-1-nitrosourea potentiates the toxicity of acetaminophen both in the phenobarbital-induced rat and in hepatocytes cultured from such animals.
- Peroxidation-dependent and peroxidation-independent mechanisms by which acetaminophen kills cultured rat hepatocytes.
- Endocytosis of superoxide dismutase is required in order for the enzyme to protect hepatocytes from the cytotoxicity of hydrogen peroxide.
- Oxidative cell injury in the killing of cultured hepatocytes by allyl alcohol.
- Toxic consequence of the abrupt depletion of glutathione in cultured rat hepatocytes.
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