Dr. Michael John Mcphaul MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
5323 Harry Hines Blvd Dallas TX, 75390About
Dr. Michael Mcphaul practices Endocrinology in Dallas, TX. Dr. Mcphaul specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Mcphaul examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Molecular defects of the androgen receptor.
- RFG (ARA70, ELE1) interacts with the human androgen receptor in a ligand-dependent fashion, but functions only weakly as a coactivator in cotransfection assays.
- Male pseudohermaphroditism caused by mutations of the human androgen receptor.
- Aromatase messenger RNA is derived from the proximal promoter of the aromatase
- The androgen receptor (AR) in syndromes of androgen insensitivity and in prostate
- Complexities of androgen action.
- Use of the probasin promoter ARR2PB to express Bax in androgen receptor-positive prostate cancer cells.
- Expression of the human androgen receptor in eukaryotic cells using a recombinant adenovirus vector yields high levels of the soluble, functional receptor protein.
- Immunoreactive AR and genetic alterations in subjects with androgen resistance and undetectable AR levels in genital skin fibroblast ligand-binding assays.
- Molecular defects of the androgen receptor.
- Virilization of the female spotted hyena cannot be explained by alterations in the amino acid sequence of the androgen receptor (AR).
- Ligand-dependent aggregation of polyglutamine-expanded androgen receptor in neuronal cells.
- Androgen receptor mutations and androgen insensitivity.
- Triiodothyronine decreases the activity of the proximal promoter (PII) of the aromatase gene in the mouse Sertoli cell line, TM4.
- [Clinical and molecular basis of androgen resistance].
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