Dr. Robert Francis Foronjy M.D.
Pulmonologist | Pulmonary Disease
450 CLARKSON AVE BROOKLYN NY, 11203About
Dr. Robert Foronjy practices Pulmonology in New York, NY. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Foronjy manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Temple Univ Sch of Med, Philadelphia Pa 1995
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Progressive adult-onset emphysema in transgenic mice expressing human MMP-1 in the lung.
- Particulate matter in cigarette smoke alters iron homeostasis to produce a biological effect.
- Role for cathepsin K in emphysema in smoke-exposed guinea pigs.
- Eosinophil and T cell markers predict functional decline in COPD patients.
- Protein phosphatase 2A regulates innate immune and proteolytic responses to cigarette smoke exposure in the lung.
- Edible Myrciaria vexator fruits: bioactive phenolics for potential COPD therapy.
- Increased matrix metalloproteinase (MMPs) levels do not predict disease severity or progression in emphysema.
- The glutathione peroxidase 1-protein tyrosine phosphatase 1B-protein phosphatase 2A axis. A key determinant of airway inflammation and alveolar destruction.
- The potential for resident lung mesenchymal stem cells to promote functional tissue regeneration: understanding microenvironmental cues.
- Phenolic-rich extract from the Costa Rican guava (Psidium friedrichsthalianum) pulp with antioxidant and anti-inflammatory activity. Potential for COPD therapy.
- Cigarette smoke activates the proto-oncogene c-src to promote airway inflammation and lung tissue destruction.
- Respiratory syncytial virus infections enhance cigarette smoke induced COPD in mice.
- Type-I interferons induce lung protease responses following respiratory syncytial virus infection via RIG-I-like receptors.
- Leukemia inhibitory factor protects the lung during respiratory syncytial viral infection.
- α1-Antitrypsin activates protein phosphatase 2A to counter lung inflammatory responses.
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