Kirk U Knowlton M.D.
Cardiologist | Cardiovascular Disease
200 W Arbor Dr San Diego CA, 92103About
Dr. Kirk Knowlton is a cardiologist practicing in San Diego, CA. Dr. Knowlton specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Knowlton also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Univ of Ut Sch of Med, Salt Lake Cty Ut 1984
University of Utah, College of Medicine, Salt Lake City, UT Medical Degree
Duke University, School of Medicine, Durham, NC Other Degree
UC San Diego, School of Medicine, San Diego, CA Other Degree
Chief of Cardiology Medical Degree
Professor of Medicine Medical Degree
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Enteroviral protease 2A cleaves dystrophin: evidence of cytoskeletal disruption in an acquired cardiomyopathy.
- The immune system in viral myocarditis: maintaining the balance.
- Inflammatory pathways and cardiac repair: the affliction of infarction.
- Enteroviral protease 2A directly cleaves dystrophin and is inhibited by a dystrophin-based substrate analogue.
- Enteroviral cardiomyopathy: bad news for the dystrophin-glycoprotein complex.
- Dissociation of sarcoglycans and the dystrophin carboxyl terminus from the sarcolemma in enteroviral cardiomyopathy.
- Nitric oxide inhibits dystrophin proteolysis by coxsackieviral protease 2A through S-nitrosylation: A protective mechanism against enteroviral cardiomyopathy.
- The immune response following myocardial infarction: a role for T-cell-mediated myocyte damage.
- Cardioselective infection with coxsackievirus B3 requires intact type I interferon signaling: implications for mortality and early viral replication.
- Apoptosis and oncosis in the early progression of left ventricular dysfunction in
- Dystrophin deficiency markedly increases enterovirus-induced cardiomyopathy: a genetic predisposition to viral heart disease.
- The suppressor of cytokine signaling-1 (SOCS1) is a novel therapeutic target for enterovirus-induced cardiac injury.
- Dystrophin disruption in enterovirus-induced myocarditis and dilated cardiomyopathy: from bench to bedside.
- Alternatively spliced soluble coxsackie-adenovirus receptors inhibit coxsackievirus infection.
- Chronic thromboembolic pulmonary hypertension.
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