Dr. Peter J Parker M.D.
Allergist and Immunologist | Allergy
414 N Camden Dr # 957 Beverly Hills CA, 90210About
Dr. Peter Parker practices Allergy and Immunology care in Beverly Hills, CA. Dr. Parker specializes in the diagnosis and treatment of asthma and other allergic diseases. Allergist-Immunologists are trained and certified to treat each patients sensitivity and response to allergens of varying severity. Dr. Parker provides several means of testing and treatment to increase immunity to potentially harmful substances.
Education and Training
Shahid Beheshti Univ- Fac Med- Teheran- Iran 1980
Board Certification
Allergy and ImmunologyAmerican Board of Allergy and ImmunologyABAI
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Detecting protein-phospholipid interactions. Epidermal growth factor-induced activation of phospholipase D1b in situ.
- Dephosphorylation of PKCdelta by protein phosphatase 2Ac and its inhibition by nucleotides.
- Calmodulin controls organization of the actin cytoskeleton via regulation of phosphatidylinositol (4,5)-bisphosphate synthesis in Saccharomyces cerevisiae.
- PKC epsilon controls the traffic of beta1 integrins in motile cells.
- Site-directed perturbation of protein kinase C- integrin interaction blocks carcinoma cell chemotaxis.
- Phosphorylation is required for PMA- and cell-cycle-induced degradation of protein kinase Cdelta.
- Protein kinase C phosphorylates ribosomal protein S6 kinase betaII and regulates its subcellular localization.
- Potentiation of protein kinase C zeta activity by 15-deoxy-delta(12,14)-prostaglandin J(2) induces an imbalance between mitogen-activated protein kinases and NF-kappa B that promotes apoptosis in macrophages.
- PKCepsilon is a permissive link in integrin-dependent IFN-gamma signalling that facilitates JAK phosphorylation of STAT1.
- Integrin-specific signaling pathways controlling focal adhesion formation and cell migration.
- Protein kinase C controls microtubule-based traffic but not proteasomal degradation of c-Met.
- Hyperosmotic-induced protein kinase N 1 activation in a vesicular compartment is dependent upon Rac1 and 3-phosphoinositide-dependent kinase 1.
- Protein kinase C phosphorylation: an introduction.
- Molecular dissection of the interaction between the small G proteins Rac1 and RhoA and protein kinase C-related kinase 1 (PRK1).
- Emerging and diverse roles of protein kinase C in immune cell signalling.
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