Dr. Mark A Brockman MD
Pediatrician
5109 New Cut Rd Louisville KY, 40214About
Dr. Mark Brockman is a pediatrician practicing in Louisville, KY. Dr. Brockman is a doctor who specializes in the health care of children. As a pedicatrician, Dr. Brockman diagnoses and treats infections, injuries, diseases and other disorders in children. Pediatricians typically work with infants, children, teenagers and young adults up to age 21. They practice medical care as well as preventative health care. Dr. Brockman can oversee and manage the physical, mental and emotional health of their patients.
Education and Training
Univ of Louisville Sch of Med, Louisville Ky 1988
University of Louisville School of Medicine 1988
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Herpes simplex virus.
- Herpes simplex virus vectors elicit durable immune responses in the presence of preexisting host immunity.
- Macrophage-derived complement component C4 can restore humoral immunity in C4-deficient mice.
- Myeloid C3 determines induction of humoral responses to peripheral herpes simplex virus infection.
- Use of a novel GFP reporter cell line to examine replication capacity of CXCR4- and CCR5-tropic HIV-1 by flow cytometry.
- Optimal long-term humoral responses to replication-defective herpes simplex virus require CD21/CD35 complement receptor expression on stromal cells.
- Rapid ex vivo isolation and long-term culture of human Th17 cells.
- Upregulation of PD-L1 on monocytes and dendritic cells by HIV-1 derived TLR ligands.
- Structural and functional constraints limit options for cytotoxic T-lymphocyte escape in the immunodominant HLA-B27-restricted epitope in human immunodeficiency virus type 1 capsid.
- Herpes simplex virus as a tool to define the role of complement in the immune response to peripheral infection.
- Gag-protease-mediated replication capacity in HIV-1 subtype C chronic infection: associations with HLA type and clinical parameters.
- Early selection in Gag by protective HLA alleles contributes to reduced HIV-1 replication capacity that may be largely compensated for in chronic infection.
- Reduced replication capacity of NL4-3 recombinant viruses encoding reverse transcriptase-integrase sequences from HIV-1 elite controllers.
- Influence of Gag-protease-mediated replication capacity on disease progression in individuals recently infected with HIV-1 subtype C.
- Progression to AIDS in South Africa is associated with both reverting and compensatory viral mutations.
Treatments
- Sinusitis
- Tonsillitis
- Attention Deficit Hyperactivity Disorder (adhd)
- Jaundice
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