Dr. Jonathan C Weissler MD
Pulmonologist | Pulmonary Disease
5323 Harry Hines Blvd Dallas TX, 75390About
Dr. Jonathan Weissler practices Pulmonology in Dallas, TX. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Weissler manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The hospitalist movement: caution lights flashing at the crossroads.
- Syndromes of severe asthma.
- BR22, a novel protein, interacts with thyroid transcription factor-1 and activates the human surfactant protein B promoter.
- Autoantigen Ro52 directly interacts with human IgG heavy chain in vivo in mammalian cells.
- Pleiomorphic adenoma gene-like-2, a zinc finger protein, transactivates the surfactant protein-C promoter.
- Advancing academic subspecialty medicine: the agenda for the future of the Association of Subspecialty Professors.
- Human alveolar macrophages inhibit receptor-mediated increases in intracellular calcium concentration in lymphocytes.
- The TTF-1/TAP26 complex differentially modulates surfactant protein-B (SP-B) and -C (SP-C) promoters in lung cells.
- PLAGL2 translocation and SP-C promoter activity--a cellular response of lung cells to hypoxia.
- Modulation of PLAGL2 transactivation activity by Ubc9 co-activation not SUMOylation.
- PLAGL2 expression-induced lung epithelium damages at bronchiolar alveolar duct junction in emphysema: bNip3- and SP-C-associated cell death/injury activity.
- Pleiomorphic adenoma gene-like 2 expression is associated with the development of lung adenocarcinoma and emphysema.
- Cytolytic human lung lymphocytes: characterization of intragranular protease content and response to interleukin-2.
- Inability of human alveolar macrophages to stimulate resting T cells correlates with decreased antigen-specific T cell-macrophage binding.
- Human pulmonary natural killer (NK) cells exhibit limited lymphokine-activated killer (LAK) activity.
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