Dr. Neil Richtand, MD, PhD
Psychiatrist | Psychiatry
3350 LA JOLLA VILLAGE DR SAN DIEGO CA, 92161About
Dr. Neil Richtand is a psychiatrist practicing in Cincinnati, OH. Dr. Richtand is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. Richtand diagnoses and treats mental illnesses. Dr. Richtand may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. Richtand treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
Education and Training
Vanderbilt Univ Sch of Med, Nashville Tn 1987
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Quantification of dopamine D3 receptor mRNA level associated with the development of amphetamine-induced behavioral sensitization in the rat brain.
- Olfactory receptor neurons express D2 dopamine receptors.
- The D3 dopamine receptor and substance dependence.
- Cloning, expression, and regulation of a glucocorticoid-induced receptor in rat brain: effect of repetitive amphetamine.
- Regression modeling of rodent locomotion data.
- 7-OH-DPAT and PD 128907 selectively activate the D3 dopamine receptor in a novel environment.
- Analysis of gene expression in striatal tissue by multiprobe RNase protection assay.
- Laboratory analysis of behavioral effects of drugs of abuse in rodents.
- Expression of the glucocorticoid-induced receptor mRNA in rat brain.
- Effects of cytochrome P450 3A modulators ketoconazole and carbamazepine on quetiapine pharmacokinetics.
- Behavioral sensitization, alternative splicing, and d3 dopamine receptor-mediated inhibitory function.
- Dose-response analysis of locomotor activity and stereotypy in dopamine D3 receptor mutant mice following acute amphetamine.
- Modulation of phosphoinositide-protein kinase C signal transduction by omega-3 fatty acids: implications for the pathophysiology and treatment of recurrent neuropsychiatric illness.
- C57BL/6J mice exhibit reduced dopamine D3 receptor-mediated locomotor-inhibitory function relative to DBA/2J mice.
- Relative expression of D3 dopamine receptor and alternative splice variant D3nf mRNA in high and low responders to novelty.
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