Dr. John Tolliver Curnutte M.D., PH.D.
Hematologist (Pediatric) | Pediatric Hematology-Oncology
901 S California Ave Palo Alto CA, 94304About
Dr. John Curnutte is a pediatric hematologist practicing in Palo Alto, CA. Dr. Curnutte specializes in treating children that have a blood disease or cancer. Such blood diseases include disorders of red blood cells, white blood cells and/or platelets. The types of cancers that Dr. Curnutte treats include leukemias, lymphomas and certain tumors. Dr. Curnutte can also treat bleeding disorders in children. Pediatric hematologists can be found in childrens hospitals, community hospitals, university medical centers and more.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Simultaneous presence of p47(phox) and flavocytochrome b-245 are required for the activation of NADPH oxidase by anionic amphiphiles. Evidence for an intermediate state of oxidase activation.
- The mechanism of activation of NADPH oxidase in the cell-free system: the activation process is primarily catalytic and not through the formation of a stoichiometric complex.
- Activation of human neutrophil NADPH oxidase by phosphatidic acid or diacylglycerol in a cell-free system. Activity of diacylglycerol is dependent on its conversion to phosphatidic acid.
- A novel mutation in the CYBB gene resulting in an unexpected pattern of exon skipping and chronic granulomatous disease.
- Molecular characterization of autosomal recessive chronic granulomatous disease caused by a defect of the nicotinamide adenine dinucleotide phosphate (reduced form) oxidase component p67-phox.
- Autosomal recessive chronic granulomatous disease caused by novel mutations in NCF-2, the gene encoding the p67-phox component of phagocyte NADPH oxidase.
- Genomic structure of the human p47-phox (NCF1) gene.
- Biological defense mechanisms. Evidence for the participation of superoxide in bacterial killing by xanthine oxidase.
- Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22(phox).
- Experimental histoplasmosis in mice treated with anti-murine interferon-gamma antibody and in interferon-gamma gene knockout mice.
- Hematologically important mutations: the autosomal recessive forms of chronic granulomatous disease (first update).
- Autosomal recessive chronic granulomatous disease caused by defects in NCF-1, the gene encoding the phagocyte p47-phox: mutations not arising in the NCF-1 pseudogenes.
- Role of C5a in multiorgan failure during sepsis.
- Hematologically important mutations: X-linked chronic granulomatous disease (second update).
- Transplantation of a fetus with paternal Thy-1(+)CD34(+)cells for chronic
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