Dr. Benjamin David Ehst M.D., PH.D.
Dermatologist
601 N Caroline St Suite 6066 Baltimore MD, 21205About
Dr. Benjamin Ehst is a dermatologist practicing in Baltimore, MD. Dr. Ehst specializes in skin care. Dermatologists evaluate and manage both common and uncommon skin conditions. These conditions include acne, psoriasis, warts, skin infections, atopic dermatitis, herpes simplex and more. Dermatologists are also experts in more complex skin diseases like impetigo, hidradenitis and milaria. Dr. Ehst diagnoses skin problems and develops unique treatments plans for each individual patient.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Bacterial flagellin is an effective adjuvant for CD4+ T cells in vivo.
- Distinct dendritic cell populations sequentially present antigen to CD4 T cells and stimulate different aspects of cell-mediated immunity.
- Development of a novel transgenic mouse for the study of interactions between CD4 and CD8 T cells during graft rejection.
- CD4+ T-cell help controls CD8+ T-cell memory via TRAIL-mediated activation-induced cell death.
- Primary cutaneous CD30+ anaplastic large cell lymphoma responds to imiquimod cream.
- Toll-like receptor 2, 4, 7 and 9 expression in primary cutaneous CD30+ T-cell lymphoma.
- Cutaneous manifestations of internal malignancy.
- Chronic ulcerative herpes simplex virus infection of the vulva.
- A double-blind, randomized, bilateral comparison of skin irritancy following application of the combination acne products clindamycin/tretinoin and benzoyl peroxide/adapalene.
- Pityriasis rubra pilaris-like eruption associated with the multikinase inhibitor ponatinib.
- Triple antibiotic combination therapy may improve but not resolve granuloma annulare.
- Unique Cutaneous Reaction to Second- and Third-Generation Tyrosine Kinase Inhibitors for Chronic Myeloid Leukemia.
- Nicolau syndrome and localized panniculitis: a report of dual diagnoses with an emphasis on morphea profunda-like changes following injection with glatiramer acetate.
- CD28/B7 interactions deliver a unique signal to naive T cells that regulates cell survival but not early proliferation.
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