Dr. Frederic Joseph Kaye MD
Hematologist-Oncologist | Hematology & Oncology
1600 Sw Archer Rd Gainesville FL, 32610About
Dr. Frederic Kaye is a hematologist oncologist practicing in Gainesville, FL. Dr. Kaye specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The 100-kDa proteolytic fragment of RB is retained predominantly within the nuclear compartment of apoptotic cells.
- Temperature-sensitive RB mutations linked to incomplete penetrance of familial retinoblastoma in 12 families.
- Closing in on another renal cancer suppressor locus near chromosome 3p14.
- The p16 status of tumor cell lines identifies small molecule inhibitors specific for cyclin-dependent kinase 4.
- Induction of Myc-intron-binding polypeptides MIBP1 and RFX1 during retinoic acid-mediated differentiation of haemopoietic cells.
- A family of ubiquitin-like proteins binds the ATPase domain of Hsp70-like Stch.
- Daily uplifts, hassles, stresses and cognitive failures: in cigarette smokers, abstaining smokers, and non-smokers.
- Disruption of Myc-tubulin interaction by hyperphosphorylation of c-Myc during mitosis or by constitutive hyperphosphorylation of mutant c-Myc in Burkitt's lymphoma.
- Assignment of ubiquilin2 (UBQLN2) to human chromosome xp11. 23-->p11.1 by GeneBridge radiation hybrids.
- Protein expression of the RB-related gene family and SV40 large T antigen in mesothelioma and lung cancer.
- Searching for selective cyclin-dependent kinase inhibitors to target the retinoblastoma/p16 cancer gene pathway.
- Molecular biology of lung cancer.
- Publishing negative data: beta-tubulin mutations in lung cancer.
- RB and cyclin dependent kinase pathways: defining a distinction between RB and p16 loss in lung cancer.
- t(11;19)(q21;p13) translocation in mucoepidermoid carcinoma creates a novel fusion product that disrupts a Notch signaling pathway.
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