Dr. Jay H Chung DDS
Dentist
10 Brent Ct Potomac MD, 20854About
Dr. Jay Chung is a Dentist practicing in Potomac, MD. Dr. Chung specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
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Expert Publications
Data provided by the National Library of Medicine- Determination of substrate motifs for human Chk1 and hCds1/Chk2 by the oriented peptide library approach.
- Human T-cell leukemia virus type 1 Tax interacts with Chk1 and attenuates DNA-damage induced G2 arrest mediated by Chk1.
- 53BP1 is associated with replication protein A and is required for RPA2 hyperphosphorylation following DNA damage.
- Promyelocytic leukemia activates Chk2 by mediating Chk2 autophosphorylation.
- ATR, PML, and CHK2 play a role in arsenic trioxide-induced apoptosis.
- Oxygen tension regulates the stability of insulin receptor substrate-1 (IRS-1) through caspase-mediated cleavage.
- Identification of novel substrates for human checkpoint kinase Chk1 and Chk2 through genome-wide screening using a consensus Chk phosphorylation motif.
- Activation of 5'-AMP-activated kinase with diabetes drug metformin induces casein kinase Iepsilon (CKIepsilon)-dependent degradation of clock protein mPer2.
- Alkylating agent methyl methanesulfonate (MMS) induces a wave of global protein hyperacetylation: implications in cancer cell death.
- Nek6 is involved in G2/M phase cell cycle arrest through DNA damage-induced phosphorylation.
- The role of mPer2 clock gene in glucocorticoid and feeding rhythms.
- CK2 is the regulator of SIRT1 substrate-binding affinity, deacetylase activity and cellular response to DNA-damage.
- AMP-activated protein kinase-deficient mice are resistant to the metabolic effects of resveratrol.
- Glucose deprivation is associated with Chk1 degradation through the ubiquitin-proteasome pathway and effective checkpoint response to replication blocks.
- AMPK regulates circadian rhythms in a tissue- and isoform-specific manner.
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