Dr. Tony T Huang MD
OB-GYN (Obstetrician-Gynecologist)
158 Linwood Plz Fort Lee NJ, 07024About
Dr. Tony Huang is an obstetrician-gynecologist practicing in Fort Lee, NJ. Dr. Huang specializes in women's health, particularly the female reproductive system, pregnancy and childbirth. As an obstetrician-gynecologist, or OB-GYN, Dr. Huang can treat a number of health issues related to the vagina, uterus, ovaries, fallopian tubes and breasts. Dr Huang can also treat women during pregnancy, labor, childbirth and the postpartum period. In this specialty, doctors focus on reproductive care from puberty through adulthood.
Education and Training
Mt Sinai Sch of Med of The City Univ of Ny, New York Ny 1987
Board Certification
Obstetrics and GynecologyAmerican Board of Obstetrics and GynecologyABOG
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The zinc finger domain of NEMO is selectively required for NF-kappa B activation by UV radiation and topoisomerase inhibitors.
- Sequential modification of NEMO/IKKgamma by SUMO-1 and ubiquitin mediates NF-kappaB activation by genotoxic stress.
- The deubiquitinating enzyme USP1 regulates the Fanconi anemia pathway.
- Regulation of DNA repair by ubiquitylation.
- HAUSP hunting the FOX(O).
- Calcium-dependent regulation of NEMO nuclear export in response to genotoxic stimuli.
- FANCI is a second monoubiquitinated member of the Fanconi anemia pathway.
- A UAF1-containing multisubunit protein complex regulates the Fanconi anemia pathway.
- Patient-derived C-terminal mutation of FANCI causes protein mislocalization and reveals putative EDGE motif function in DNA repair.
- Nuclear export of the NF-κB inhibitor IκBα is required for proper B cell and secondary lymphoid tissue formation.
- APC/CCdh1-dependent proteolysis of USP1 regulates the response to UV-mediated DNA damage.
- Insights into phosphorylation-dependent mechanisms regulating USP1 protein stability during the cell cycle.
- Dysregulation of DNA polymerase κ recruitment to replication forks results in genomic instability.
- The Fanconi anemia pathway in replication stress and DNA crosslink repair.
- The auto-generated fragment of the Usp1 deubiquitylase is a physiological substrate of the N-end rule pathway.
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