Dr. Kendall Alan Crutcher MD
Anesthesiologist
2408 E 81ST ST TULSA OK, 74137About
Dr. Kendall Crutcher is an anesthesiologist practicing in Newport, RI. Dr. Crutcher ensures the safety of patients who are about to undergo surgery. Anestesiologists specialize in general anesthesia, which will (put the patient to sleep), sedation, which will calm the patient or make him or her unaware of the situation, and regional anesthesia, which just numbs a specific part of the body. As an anesthesiologist, Dr. Crutcher also might help manage pain after an operation.
Board Certification
AnesthesiologyAmerican Board of AnesthesiologyABA
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Enhanced neurotrophin-induced axon growth in myelinated portions of the CNS in mice lacking the p75 neurotrophin receptor.
- Truncated apolipoprotein E (ApoE) causes increased intracellular calcium and may mediate ApoE neurotoxicity.
- White matter of the CNS supports or inhibits neurite outgrowth in vitro depending on geometry.
- NGF expression in the aged rat pineal gland does not correlate with loss of sympathetic axonal branches and varicosities.
- The role of NGF in pregnancy-induced degeneration and regeneration of sympathetic nerves in the guinea pig uterus.
- Remodeling of adult sensory axons in the superior cervical ganglion in response to exogenous nerve growth factor.
- Sympathetic neurite growth on central nervous system sections is region-specific and unaltered by aging.
- Absence of p75(NTR) expression reduces nerve growth factor immunolocalization in cholinergic septal neurons.
- Disruption of spinal cord white matter and sciatic nerve geometry inhibits axonal growth in vitro in the absence of glial scarring.
- Myelin contributes to the parallel orientation of axonal growth on white matter in vitro.
- Neuronal-vascular relationships in the raphe nuclei, locus coeruleus, and substantia nigra in primates.
- Neuronal-vascular relationships in the raphe nuclei, locus coeruleus, and substantia nigra in primates.
- Evidence for reduced accumulation of exogenous neurotrophin by aged sympathetic neurons.
- Reduced age-related plasticity of neurotrophin receptor expression in selected sympathetic neurons of the rat.
- Nerve growth factor-induced sprouting of mature, uninjured sympathetic axons.
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