Dr. Donald Robert Spitz M.D.
Family Practitioner
219 Via Lido Soud Newport Beach CA, 92663About
Dr. Donald Spitz is a family practitioner practicing in Newport Beach, CA. Dr. Spitz specializes in comprehensive health care for people of all ages. In addition to diagnosing and treating illnesses, family practitioners also put focus on preventative care with routine checkups, tests and personalized coaching on how to maintain a healthy lifestyle. Dr. Spitz possesses immense general knowledge on maintaining health and today, family practitioners provide more care for the underserved and rural populations than any other medical specialty.
Education and Training
Chicago Medical School at Rosalind Franklin University of Medicine & Science 1956
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Redox factor-1 (Ref-1) mediates the activation of AP-1 in HeLa and NIH 3T3 cells in response to heat shock.
- Radiofrequency electromagnetic fields have no effect on the in vivo proliferation of the 9L brain tumor.
- Dominant-negative Jun N-terminal protein kinase (JNK-1) inhibits metabolic oxidative stress during glucose deprivation in a human breast carcinoma cell line.
- Glucose deprivation-induced oxidative stress in human tumor cells. A fundamental defect in metabolism?
- A spectrophotometric method for the direct detection and quantitation of nitric oxide, nitrite, and nitrate in cell culture media.
- A role for oxidative stress in suppressing serum immunoglobulin levels in lead-exposed Fisher 344 rats.
- Glial cell type-specific responses to menadione-induced oxidative stress.
- Cell cycle-coupled variation in topoisomerase IIalpha mRNA is regulated by the 3'-untranslated region. Possible role of redox-sensitive protein binding in mRNA accumulation.
- An alternative method to stereotactic inoculation of transplantable brain tumours in large numbers of rats.
- Cytometric methods to analyze thermal effects.
- Thioredoxin nuclear translocation and interaction with redox factor-1 activates the activator protein-1 transcription factor in response to ionizing radiation.
- Increased activator protein 1 activity as well as resistance to heat-induced radiosensitization, hydrogen peroxide, and cisplatin are inhibited by indomethacin in oxidative stress-resistant cells.
- H(2)O(2)-induced O(2) production by a non-phagocytic NAD(P)H oxidase causes oxidant injury.
- Redox modulation of the pro-fibrogenic mediator plasminogen activator inhibitor-1 following ionizing radiation.
- The effect of monosaturated and polyunsaturated fatty acids on oxygen toxicity in cultured cells.
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