Dr. Samir Mukund Parikh M.D.
Nephrologist (Kidney Specialist) | Nephrology
330 Brookline Ave Rw 563 Boston MA, 02215About
Dr. Samir Parikh is a nephrologist practicing in Boston, MA. Dr. Parikh specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Parikh most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Vanderbilt University School of Medicine 2001
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Nephrology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The nature of the autonomic dysfunction in multiple system atrophy.
- Images in clinical medicine. Calcinosis associated with renal failure.
- Angiopoietin 2 is a partial agonist/antagonist of Tie2 signaling in the endothelium.
- Effects of a synthetic PEG-ylated Tie-2 agonist peptide on endotoxemic lung injury and mortality.
- PGC-1α promotes recovery after acute kidney injury during systemic inflammation in mice.
- Angiopoietin-1 requires IQ domain GTPase-activating protein 1 to activate Rac1 and promote endothelial barrier defense.
- Circulating mediators of focal segmental glomerulosclerosis: soluble urokinase plasminogen activator receptor in context.
- The synthetic tie2 agonist peptide vasculotide protects against vascular leakage and reduces mortality in murine abdominal sepsis.
- Vascular endothelial hyperpermeability induces the clinical symptoms of Clarkson disease (the systemic capillary leak syndrome).
- Impaired function of the Tie-2 receptor contributes to vascular leakage and lethality in anthrax.
- Angiopoietin-2 is a potential mediator of endothelial barrier dysfunction following cardiopulmonary bypass.
- Angiopoietin-2 may contribute to multiple organ dysfunction and death in sepsis*.
- Mending leaky blood vessels: the angiopoietin-Tie2 pathway in sepsis.
- Dysregulation of the angiopoietin-Tie-2 axis in sepsis and ARDS.
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