Dr. Robert Leo Fairchild MD
Anesthesiologist | Pain Medicine
6801 Emmett F Lowry Expy Texas City TX, 77591About
Dr. Robert Fairchild is an anesthesiologist practicing in Texas City, TX. Dr. Fairchild ensures the safety of patients who are about to undergo surgery. Anestesiologists specialize in general anesthesia, which will (put the patient to sleep), sedation, which will calm the patient or make him or her unaware of the situation, and regional anesthesia, which just numbs a specific part of the body. As an anesthesiologist, Dr. Fairchild also might help manage pain after an operation.
Education and Training
Univ of Tx Med Sch At San Antonio, San Antonio Tx 1984
Board Certification
AnesthesiologyAmerican Board of AnesthesiologyABA
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Skin allograft rejection is suppressed in mice lacking the antiviral enzyme, 2',5'-oligoadenylate-dependent RNase L.
- Chronic antagonism of Mig inhibits cellular infiltration and promotes survival of class II MHC disparate skin allografts.
- Chemokines: directing leukocyte infiltration into allografts.
- Chemokine and chemokine receptor gene expression indicates acute rejection of human cardiac transplants.
- Neutrophils mediate parenchymal tissue necrosis and accelerate the rejection of complete major histocompatibility complex-disparate cardiac allografts in the absence of interferon-gamma.
- T-cell mediated induction of allogeneic endothelial cell chemokine expression.
- Absence of allograft ICAM-1 attenuates alloantigen-specific T cell priming, but not primed T cell trafficking into the graft, to mediate acute rejection.
- Chemokine and receptor-gene expression during early and late acute rejection episodes in human cardiac allografts.
- The Yin and Yang of IFN-gamma in allograft rejection.
- Effects of T cell frequency and graft size on transplant outcome in mice.
- CD4+ T cells regulate CD8+ T cell-mediated cutaneous immune responses by restricting effector T cell development through a Fas ligand-dependent mechanism.
- CD40 engagement enhances antigen-presenting langerhans cell priming of IFN-gamma-producing CD4+ and CD8+ T cells independently of IL-12.
- The intensity of neutrophil infiltration controls the number of antigen-primed CD8 T cells recruited into cutaneous antigen challenge sites.
- Act1, a negative regulator in CD40- and BAFF-mediated B cell survival.
- Alloreactive T cell responses and acute rejection of single class II MHC-disparate heart allografts are under strict regulation by CD4+ CD25+ T cells.
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