Dr. Michael A. Resnick D.O.
Gastroenterologist | Gastroenterology
9501 Roosevelt Blvd Suite 103 Philadelphia PA, 19114Rating
4/5
About
Dr. Michael Resnick is a gastroenterologist practicing in Philadelphia, PA. Dr. Resnick specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Resnick performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Education and Training
Philadelphia Coll of Osteo Med, Philadelphia Pa 1977
Pennsylvania State University College of Medicine 1977
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Differential suppression of DNA repair deficiencies of Yeast rad50, mre11 and xrs2 mutants by EXO1 and TLC1 (the RNA component of telomerase).
- The Mre11 complex is required for repair of hairpin-capped double-strand breaks and prevention of chromosome rearrangements.
- Tumour p53 mutations exhibit promoter selective dominance over wild type p53.
- The mitochondrial protein frataxin prevents nuclear damage.
- Fidelity of DNA polymerase epsilon holoenzyme from budding yeast Saccharomyces cerevisiae.
- A novel p53 mutational hotspot in skin tumors from UV-irradiated Xpc mutant mice alters transactivation functions.
- The flexible loop of human FEN1 endonuclease is required for flap cleavage during DNA replication and repair.
- Okazaki fragment maturation in yeast. II. Cooperation between the polymerase and 3'-5'-exonuclease activities of Pol delta in the creation of a ligatable nick.
- Differential transactivation by the p53 transcription factor is highly dependent on p53 level and promoter target sequence.
- Cadmium is a mutagen that acts by inhibiting mismatch repair.
- Functional mutants of the sequence-specific transcription factor p53 and implications for master genes of diversity.
- Reduction in frataxin causes progressive accumulation of mitochondrial damage.
- Chromosomal site-specific double-strand breaks are efficiently targeted for repair by oligonucleotides in yeast.
- Cell cycle progression in G1 and S phases is CCR4 dependent following ionizing radiation or replication stress in Saccharomyces cerevisiae.
- Role of the nuclease activity of Saccharomyces cerevisiae Mre11 in repair of DNA double-strand breaks in mitotic cells.
Treatments
- Pain
Dr. Michael A. Resnick D.O.'s Practice location
Practice At 9501 Roosevelt Blvd Suite 103
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Nearest Hospitals
NAZARETH HOSPITALl
2601 HOLME AVE PHILADELPHIA PA 19152ARIA HEALTHl
10800 KNIGHTS ROAD PHILADELPHIA PA 19114