Dr. Mason Wright Freeman MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
55 Fruit Street Grj 1321 Lipid Metabolism Uni Boston MA, 02114Rating
5/5
About
Dr. Mason Freeman practices Endocrinology in Boston, MA. Dr. Freeman specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Freeman examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ of Ca, San Francisco, Sch of Med, San Francisco Ca 1979
University of California 1979
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- C/EBPalpha induces adipogenesis through PPARgamma: a unified pathway.
- Naturally occurring mutations in the largest extracellular loops of ABCA1 can disrupt its direct interaction with apolipoprotein A-I.
- A CD36-initiated signaling cascade mediates inflammatory effects of beta-amyloid.
- Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophages.
- Activation of signaling pathways by putative scavenger receptor class A (SR-A) ligands requires CD14 but not SR-A.
- Cross-linking and lipid efflux properties of apoA-I mutants suggest direct association between apoA-I helices and ABCA1.
- Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways.
- ATP-binding cassette transporter A1 contains a novel C-terminal VFVNFA motif that is required for its cholesterol efflux and ApoA-I binding activities.
- The induction of macrophage gene expression by LPS predominantly utilizes Myd88-independent signaling cascades.
- Abca7 null mice retain normal macrophage phosphatidylcholine and cholesterol efflux activity despite alterations in adipose mass and serum cholesterol levels.
- Requirement of JNK2 for scavenger receptor A-mediated foam cell formation in atherogenesis.
- Atherosclerosis and innate immune signaling.
- Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic mice.
- Nonparametric methods for microarray data based on exchangeability and borrowed power.
Treatments
- High Cholesterol
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Dr. Mason Freeman, MD
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Nearest Hospitals
MASSACHUSETTS EYE AND EAR INFIRMARYl
243 CHARLES STREET BOSTON MA 2114SHRINERS' HOSPITAL FOR CHILDREN - BOSTON, THEl
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800 WASHINGTON STREET BOSTON MA 2111