Dr. Linda Kathyn Bockenstedt MD
Rheumatologist | Rheumatology
789 Howard Ave Dana Building - 3rd New Haven CT, 06519About
Dr. Linda Bockenstedt is a rheumatologist practicing in New Haven, CT. Dr. Bockenstedt specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Bockenstedt treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Education and Training
Ohio State University College of Medicine 1981
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Rheumatology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- T cell autoimmunity in Ig transgenic mice.
- Cutting edge: CD1d deficiency impairs murine host defense against the spirochete, Borrelia burgdorferi.
- CD4+ T helper 1 cells facilitate regression of murine Lyme carditis.
- Natural antibody affects survival of the spirochete Borrelia burgdorferi within feeding ticks.
- Detection of attenuated, noninfectious spirochetes in Borrelia burgdorferi-infected mice after antibiotic treatment.
- Humoral immunity reflects altered T helper cell bias in Borrelia burgdorferi-infected gamma delta T-cell-deficient mice.
- OspC facilitates Borrelia burgdorferi invasion of Ixodes scapularis salivary glands.
- Circulating fibrocytes: collagen-secreting cells of the peripheral blood.
- Myeloid differentiation antigen 88 deficiency impairs pathogen clearance but does not alter inflammation in Borrelia burgdorferi-infected mice.
- Infection-induced marginal zone B cell production of Borrelia hermsii-specific antibody is impaired in the absence of CD1d.
- MyD88 deficiency enhances acquisition and transmission of Borrelia burgdorferi by Ixodes scapularis ticks.
- Marginal zone B-cell depletion impairs murine host defense against Borrelia burgdorferi infection.
- IL-12/23p40-dependent clearance of Anaplasma phagocytophilum in the murine model of human anaplasmosis.
- Signaling via the inositol phospholipid pathway by T cell antigen receptor is limited by receptor number.
- Langerhans cell deficiency impairs Ixodes scapularis suppression of Th1 responses in mice.
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