Dr. Charles William Heilig MD
Nephrologist (Kidney Specialist) | Nephrology
655 W 8th St Ufjp Nephrology Jacksonville FL, 32209About
Dr. Charles Heilig is a nephrologist practicing in Jacksonville, FL. Dr. Heilig specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Heilig most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
University of Minnesota Medical School 1984
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Nephrology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Glucose transporters control gene expression of aldose reductase, PKCalpha, and GLUT1 in mesangial cells in vitro.
- Glucose-specific regulation of aldose reductase in human retinal pigment epithelial cells in vitro.
- Identification of a novel form of renal glucosuria with overexcretion of arginine, carnosine, and taurine.
- Evidence for a novel TGF-beta1-independent mechanism of fibronectin production in mesangial cells overexpressing glucose transporters.
- Regulation of cytosolic pH and lactic acid release in mesangial cells overexpressing GLUT1.
- Mouse models of diabetic nephropathy.
- Glucose transporters in diabetic nephropathy.
- Diabetic nephropathy: of mice and men.
- Increased glucose uptake and metabolism in mesangial cells overexpressing glucose transporter 1 increases interleukin-6 and vascular endothelial growth factor production: role of AP-1 and HIF-1alpha.
- Nephron-deficient Fvb mice develop rapidly progressive renal failure and heavy albuminuria involving excess glomerular GLUT1 and VEGF.
- Upregulation of inositol transport mediates inositol accumulation in hyperosmolar brain cells.
- Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis.
- Methylamine and polyol responses to salt loading in renal inner medulla.
- Organic osmolytes in inner medulla of Brattleboro rat: effects of ADH and dehydration.
- A Unique Cause of Proteinuria in Pregnancy: Class II Lupus Nephritis with Concomitant Minimal Change Disease.
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