Dr. Kenneth S Polonsky MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
660 S Euclid Ave Saint Louis MO, 63110About
Dr. Kenneth Polonsky practices Endocrinology in Saint Louis, MO. Dr. Polonsky specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Polonsky examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ Of The Witwatersrand- Med Sch- Johannesburg- So Africa 1973
University of The Witwatersrand, Faculty of Health Sciences,Parktown, South Africa 1973
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Increased beta-cell proliferation and reduced mass before diabetes onset in the nonobese diabetic mouse.
- Basal insulin hypersecretion in insulin-resistant Zucker diabetic and Zucker fatty rats: role of enhanced fuel metabolism.
- Metabolic effects of short-term elevations of plasma cortisol are more pronounced in the evening than in the morning.
- Evolution of beta-cell dysfunction in impaired glucose tolerance and diabetes.
- Overexpression of Bcl-x(L) in beta-cells prevents cell death but impairs mitochondrial signal for insulin secretion.
- Roles of insulin resistance and obesity in regulation of plasma insulin
- Failure of physiological plasma glucose excursions to entrain high-frequency pulsatile insulin secretion in type 2 diabetes.
- Interactions between insulin resistance and insulin secretion in the development of glucose intolerance.
- Twenty-four-hour leptin levels respond to cumulative short-term energy imbalance and predict subsequent intake.
- Dynamics of insulin secretion in obesity and diabetes.
- Quantitative indexes of beta-cell function during graded up&down glucose infusion from C-peptide minimal models.
- Glucagon-like peptide-1 stimulates insulin secretion by a Ca2+-independent mechanism in Zucker diabetic fatty rat islets of Langerhans.
- Oral glucose tolerance test minimal model indexes of beta-cell function and insulin sensitivity.
- GLP-1-induced alterations in the glucose-stimulated insulin secretory dose-response curve.
- Calpains play a role in insulin secretion and action.
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