Lili A Barouch M.D.
Cardiologist | Cardiovascular Disease
4940 Eastern Ave Baltimore MD, 21224About
Dr. Lili Barouch is a cardiologist practicing in Baltimore, MD. Dr. Barouch specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Barouch also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Johns Hopkins Univ Sch of Med, Baltimore Md 1996
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Leptin signaling and obesity: cardiovascular consequences.
- Decreased p110alpha catalytic activity accompanies increased myocyte apoptosis
- Cardiomyocyte apoptosis in animal models of obesity.
- Resolution of giant cell myocarditis after extended ventricular assistance.
- Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the beta3-adrenoreceptor.
- Leptin is essential in maintaining normal vascular compliance independent of body weight.
- Effect of intermittent hypoxia on atherosclerosis in apolipoprotein E-deficient mice.
- PDE5A suppression of acute beta-adrenergic activation requires modulation of myocyte beta-3 signaling coupled to PKG-mediated troponin I phosphorylation.
- Beta 3-adrenoreceptor regulation of nitric oxide in the cardiovascular system.
- Restoring leptin signaling reduces hyperlipidemia and improves vascular stiffness induced by chronic intermittent hypoxia.
- Exercise protects against myocardial ischemia-reperfusion injury via stimulation of β(3)-adrenergic receptors and increased nitric oxide signaling: role of nitrite and nitrosothiols.
- Dependence of β3-adrenergic signaling on the adipokine leptin in cardiac myocytes.
- Cardioprotective effect of beta-3 adrenergic receptor agonism: role of neuronal nitric oxide synthase.
- Interleukin 10 knockout frail mice develop cardiac and vascular dysfunction with increased age.
- Beneficial cardiac effects of caloric restriction are lost with age in a murine model of obesity.
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