Dr. Michael Karl Racke, MD
Nephrologist (Kidney Specialist) | Neurology
134 Mendolin Way Powell OH, 43065About
Dr Michael K. Racke is Medical Director for Neurology at Quest Diagnostics. Dr Racke is a leader in the field of neurology and neuroimmunology and author of more than 200 peer-reviewed papers, book chapters, and reviews on the pathogenesis of neuroimmunologic diseases. He has over 3 decades of leadership experience in academia and clinical practice, and continues to serve on several national professional society committees.
Education and Training
Umdnj-New Jersey Med Sch- Newark Nj 1985
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Blockade of CD28 during in vitro activation of encephalitogenic T cells or after disease onset ameliorates experimental autoimmune encephalomyelitis.
- Differential requirements of naïve and memory T cells for CD28 costimulation in autoimmune pathogenesis.
- Age dependence of clinical and pathological manifestations of autoimmune demyelination. Implications for multiple sclerosis.
- T cells are the main cell type expressing B7-1 and B7-2 in the central nervous system during acute, relapsing and chronic experimental autoimmune encephalomyelitis.
- The role of CTLA-4 in tolerance induction and T cell differentiation in experimental autoimmune encephalomyelitis: i.p. antigen administration.
- The role of CTLA-4 in tolerance induction and ttigen administration cell differentiation in experimental autoimmune encephalomyelitis: i. v. antigen administration.
- Retrovirus mediated gene transfer of the self antigen MBP into the bone marrow of mice alters resistance to experimental autoimmune encephalomyelitis.
- The role of costimulation in autoimmune demyelination.
- Developmentally regulated gene expression of Th2 cytokines in the brain.
- Role of nerve growth factor in experimental autoimmune encephalomyelitis.
- Ocular contrapulsion in multiple sclerosis: clinical features and pathophysiological mechanisms.
- Autonomic regulation of neuroimmunological responses: implications for multiple
- T cell epitopes of human myelin oligodendrocyte glycoprotein identified in HLA-DR4 (DRB1*0401) transgenic mice are encephalitogenic and are presented by human B cells.
- Peroxisome proliferator-activated receptor-gamma agonist 15-deoxy-Delta(12,14)-prostaglandin J(2) ameliorates experimental autoimmune encephalomyelitis.
- Glatiramer acetate (Copaxone) therapy induces CD8(+) T cell responses in patients with multiple sclerosis.
Treatments
- Neuroimmunology, Multiple Sclerosis (ms), Myelitis And More
Professional Memberships
- American Academy of Neurology
Fellowships
- Neuroimmunology: National Institutes of Health, NCCAM 1991
- National Institute of Allergy and Infectious Disease 1989
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Nearby Providers
- Dr. Ranjana Sinha M.D.10330 SAWMILL PARKWAY, SUITE # 202 POWELL OH 43065