Dr. Paul Cyril Van ness MD
Neurologist | Neurology
5323 Harry Hines Blvd Dallas TX, 75390About
Dr. Paul Van ness is a distinguished Neurologist in Dallas, TX. Dr. Van ness specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Van ness employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Van ness is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Univ of Ca, Los Angeles, Ucla Sch of Med, Los Angeles Ca 1981
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Nonconvulsive status epilepticus resulting from Jarisch-Herxheimer reaction in a patient with neurosyphilis.
- Neurophysiologic and neuroradiologic features of intractable epilepsy after traumatic brain injury in adults.
- The concept of risk in biomedical research involving human subjects.
- Therapy for the epilepsies.
- Evolving treatment strategies for epilepsy.
- Increased risk of late posttraumatic seizures associated with inheritance of APOE
- Evaluation of seizure-like episodes in survivors of moderate and severe traumatic brain injury.
- The clinical significance of EEG cyclic alternating pattern during coma.
- Posttraumatic epilepsy: the endophenotypes of a human model of epileptogenesis.
- Surgery is the best option for intractable unilateral mesial temporal epilepsy.
- Dihydropicrotoxinin binding sites in rat brain: comparison to GABA receptors.
- Dihydropicrotoxinin binding sites in rat brain: comparison to GABA receptors.
- Quality improvement in neurology: AAN epilepsy quality measures: Report of the Quality Measurement and Reporting Subcommittee of the American Academy of Neurology.
- Gamma-aminobutyric acid binding in mammalian brain: receptor-like specificity of sodium-independent sites.
- Gamma-aminobutyric acid binding in mammalian brain: receptor-like specificity of sodium-independent sites.
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