Dr. Richard G. Gay M.D.
Doctor
19260 Sw 65th Ave Suite 420 Tualatin OR, 97062About
Dr. Richard G. Gay M.D. is a top Doctor in Tualatin, OR. With a passion for the field and an unwavering commitment to their specialty, Dr. Richard G. Gay M.D. is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Dr. Richard G. Gay M.D. is a prime example of a true leader in health care. As a leader and expert in their field, Dr. Richard G. Gay M.D. is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In Tualatin, OR, Dr. Richard G. Gay M.D. is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Chronic propranolol treatment promotes left ventricular dilation without altering systolic function after large myocardial infarction in rats.
- Captopril reduces left ventricular enlargement induced by chronic volume overload.
- Early and late effects of captopril treatment after large myocardial infarction in rats.
- Selective vasopressin inhibition in rats with heart failure decreases afterload and results in venodilatation.
- Atrial natriuretic peptide reverses angiotensin-induced venoconstriction in dogs.
- Cardiac hypertrophy induced by thyroid hormone is independent of loading conditions and beta adrenoceptor blockade.
- Importance of venodilatation in prevention of left ventricular dilatation after
- Beta-2 adrenoceptor control of the venous circulation in intact dogs.
- Atrial natriuretic peptide levels during development of chronic heart failure after myocardial infarction in rats.
- Cardiac catheterization for patients with postinfarction angina.
- Effects of 10- to 12-day treatment with L-thyroxine in rats with myocardial infarction.
- Use of acetylcholine to measure total vascular pressure-volume relationship in dogs.
- Serial changes in left ventricular relaxation and chamber stiffness after large myocardial infarction in rats.
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