Dr. Johnny Salameh Salameh M.D.
Neurologist | Neurology
2525 W UNIVERSITY AVE MUNCIE IN, 47303About
Dr. Johnny Salameh is a distinguished Neurologist in Worcester, MA. Dr. Salameh specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Salameh employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Salameh is committed to staying abreast of the latest developments in neurological research and therapies.
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN- Neuromuscular Medicine
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Patient with amyloidoma of the ulnar nerve and salivary glands.
- A 62-year-old woman with cerebral artery air embolism during commercial air travel.
- Role of activity-dependent conduction block in the diagnosis of primary demyelinating polyneuropathy.
- Phosphoglycerate mutase deficiency with tubular aggregates in a patient from Panama.
- Brachial amyotrophic diplegia in the setting of complete HIV viral load suppression.
- Vertebral artery dissection causing an acute C5 radiculopathy.
- Amyotrophic lateral sclerosis: analysis of ALS cases in a predominantly admixed population of Ecuador.
- Partial loss of TDP-43 function causes phenotypes of amyotrophic lateral sclerosis.
- Peripheral neuropathy after hair dye exposure: a case report.
- Identifying diagnostic DNA methylation profiles for facioscapulohumeral muscular dystrophy in blood and saliva using bisulfite sequencing.
- Necrotizing myopathies: an update.
- Pearls & Oy-sters: An isolated cranial nerve 6 palsy as a presentation of polycythemia vera.
- Human C9ORF72 Hexanucleotide Expansion Reproduces RNA Foci and Dipeptide Repeat Proteins but Not Neurodegeneration in BAC Transgenic Mice.
- A dystonia-like movement disorder with brain and spinal neuronal defects is caused by mutation of the mouse laminin β1 subunit, Lamb1.
- ALS-linked protein disulfide isomerase variants cause motor dysfunction.
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