Dr. Michael A. Vogelbaum M.D.
Neurosurgeon
12902 USF MAGNOLIA DR TAMPA FL, 33612About
Dr. Michael Vogelbaum practices Neurological Surgery in Cleveland, OH. As a Neurological Surgeon, Dr. Vogelbaum prevents, diagnoses, evaluates, and treats disorders of the autonomic, peripheral, and central nervous systems. Neurological Surgeons are trained to treat such disorders as spinal canal stenosis, herniated discs, tumors, fractures, and spinal deformities, among many others.
Education and Training
Univ of Va Sch of Med, Charlottesville Va 1992
Board Certification
Neurological SurgeryAmerican Board of Neurological SurgeryABNS
Provider Details
Expert Publications
Data provided by the National Library of Medicine- IL-13R(alpha)2, a decoy receptor for IL-13 acts as an inhibitor of IL-4-dependent signal transduction in glioblastoma cells.
- Inhibition of constitutively active Stat3 suppresses proliferation and induces apoptosis in glioblastoma multiforme cells.
- Posterior fossa metastases: aggressive treatment improves survival.
- Epidermal growth factor receptor antagonists: novel therapy for the treatment of high-grade gliomas.
- Temporal chimeras produced by hypothalamic transplants.
- Brain metastases.
- Aberrant Stat3 signaling by interleukin-4 in malignant glioma cells: involvement of IL-13Ralpha2.
- Convection enhanced delivery for the treatment of malignant gliomas: symposium
- S100beta as a predictor of brain metastases: brain versus cerebrovascular damage.
- Intracranial collision tumor mimicking an octreotide-SPECT positive and FDG-PET negative meningioma.
- Results of a survey of neurosurgical practice patterns regarding the prophylactic use of anti-epilepsy drugs in patients with brain tumors.
- Mapping prosody: correlation of functional magnetic resonance imaging with intraoperative electrocorticography recordings in a patient with a right-sided temporooccipital glioma. Case illustration.
- Convection enhanced delivery for treating brain tumors and selected neurological disorders: symposium review.
- Aberrant constitutive activation of nuclear factor kappaB in glioblastoma multiforme drives invasive phenotype.
- PTPmu suppresses glioma cell migration and dispersal.
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