Dr. Harris A Gelbard MD
Neurologist | Neurology with Special Qualifications in Child Neurology
601 Elmwood Ave Box 278984 Rochester NY, 14642About
Dr. Harris Gelbard is a distinguished Neurologist in Rochester, NY. Dr. Gelbard specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Gelbard employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Gelbard is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Northwestern Uniiversity 1983
Northwestern University The Feinberg School of Medicine 1983
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Paraquat elicited neurobehavioral syndrome caused by dopaminergic neuron loss.
- HIV-1-induced neuronal injury in the developing brain.
- Proteasome blockers inhibit TNF-alpha release by lipopolysaccharide stimulated macrophages and microglia: implications for HIV-1 dementia.
- HIV-1 Tat-mediated activation of glycogen synthase kinase-3beta contributes to Tat-mediated neurotoxicity.
- Expression of caspase-3 in brains from paediatric patients with HIV-1 encephalitis.
- Neuronal fractalkine expression in HIV-1 encephalitis: roles for macrophage recruitment and neuroprotection in the central nervous system.
- Functional interplay between nuclear factor-kappaB and c-Jun integrated by coactivator p300 determines the survival of nerve growth factor-dependent PC12 cells.
- Neuroprotective strategies for HIV-1-associated neurologic disease.
- Release of the neuronal glycoprotein ICAM-5 in serum after hypoxic-ischemic injury.
- Activation of glycogen synthase kinase 3 beta (GSK-3beta) by platelet activating factor mediates migration and cell death in cerebellar granule neurons.
- Neurotrophins prevent HIV Tat-induced neuronal apoptosis via a nuclear factor-kappaB (NF-kappaB)-dependent mechanism.
- Tumor necrosis factor-alpha in normal and diseased brain: Conflicting effects via intraneuronal receptor crosstalk?
- Reduced expression of glutamate transporter EAAT2 and impaired glutamate transport in human primary astrocytes exposed to HIV-1 or gp120.
- Cytokines and arachidonic metabolites produced during human immunodeficiency virus (HIV)-infected macrophage-astroglia interactions: implications for the neuropathogenesis of HIV disease.
- Synapses and Sisyphus: life without paraplegin.
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