David Chester Sane MD
Cardiologist | Cardiovascular Disease
127 Mcclanahan St Sw Suite 300 Roanoke VA, 24014About
Dr. David Sane is a cardiologist practicing in Roanoke, VA. Dr. Sane specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Sane also practices preventative medicine, helping patients maintain a heart-healthy life.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Vitaxin, a humanized monoclonal antibody to the vitronectin receptor (alphavbeta3), reduces neointimal hyperplasia and total vessel area after balloon injury in hypercholesterolemic rabbits.
- Angiostatin binds to smooth muscle cells in the coronary artery and inhibits smooth muscle cell proliferation and migration In vitro.
- Matrix Gla protein synthesis and gamma-carboxylation in the aortic vessel wall and proliferating vascular smooth muscle cells--a cell system which resembles the system in bone cells.
- A coumarin-based prodrug strategy to improve the oral absorption of RGD peptidomimetics.
- Cardiovascular implications of the factor V Leiden mutation.
- Modulation of the binding of matrix Gla protein (MGP) to bone morphogenetic protein-2 (BMP-2).
- Localization of tissue transglutaminase in human carotid and coronary artery atherosclerosis: implications for plaque stability and progression.
- Lepirudin as a safe alternative for effective anticoagulation in patients with known heparin-induced thrombocytopenia undergoing percutaneous coronary intervention: case reports.
- Arterial calcification: a review of mechanisms, animal models, and the prospects for therapy.
- Growth hormone reverses age-related cardiac myofilament dysfunction in rats.
- Growth hormone increases regional coronary blood flow and capillary density in aged rats.
- Upregulation of functional beta(3)-adrenergic receptor in the failing canine myocardium.
- Platelet aggregability in syndrome X: a role for adenosine.
- A molecular mechanism for genetic warfarin resistance in the rat.
- Reduced inhibition by abciximab in platelets with the PlA2 polymorphism.
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