Dr. Clay B. Marsh MD
Pulmonologist | Pulmonary Disease
2050 Kenny Rd Suite 2200 Columbus OH, 43221About
Dr. Clay Marsh practices Pulmonology in Columbus, OH. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Marsh manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Opposing effect by cytokines on Fas-mediated apoptosis in A549 lung epithelial cells.
- Monocyte survival factors induce Akt activation and suppress caspase-3.
- Src homology 2 domain-containing inositol polyphosphate phosphatase regulates NF-kappa B-mediated gene transcription by phagocytic Fc gamma Rs in human myeloid cells.
- Macrophage-colony-stimulating factor-induced activation of extracellular-regulated kinase involves phosphatidylinositol 3-kinase and reactive oxygen species in human monocytes.
- Presentation of nitric oxide regulates monocyte survival through effects on caspase-9 and caspase-3 activation.
- The acute respiratory distress syndrome: a role for transforming growth factor-beta 1.
- SHIP-2 inositol phosphatase is inducibly expressed in human monocytes and serves to regulate Fcgamma receptor-mediated signaling.
- TGF-beta 1 suppresses [correction of supresses] myeloid Fc gamma receptor function by regulating the expression and function of the common gamma-subunit.
- The protein-tyrosine phosphatase SHP-1 associates with the phosphorylated immunoreceptor tyrosine-based activation motif of Fc gamma RIIa to modulate signaling events in myeloid cells.
- The inositol 5'-phosphatase SHIP-1 and the Src kinase Lyn negatively regulate macrophage colony-stimulating factor-induced Akt activity.
- M-CSF induces vascular endothelial growth factor production and angiogenic activity from human monocytes.
- Mac-1 and Fas activities are concurrently required for execution of smooth muscle cell death by M-CSF-stimulated macrophages.
- The role of ROS and RNS in regulating life and death of blood monocytes.
- The inositol 3-phosphatase PTEN negatively regulates Fc gamma receptor signaling, but supports Toll-like receptor 4 signaling in murine peritoneal macrophages.
- Proteome comparison of alveolar macrophages with monocytes reveals distinct protein characteristics.
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