Dr. Timothy Junius Triche MD, PHD
Pathologist (Pediatric) | Pediatric Pathology
4650 W Sunset Blvd Los Angeles CA, 90027About
Dr. Timothy Triche practices Pediatric Pathology in Los Angeles, CA. As a pediatric pathologist, Dr. Triche is skilled in the diagnosis and characterization of neoplastic and non-neoplastic diseases of children. Pediatric pathologists gain expertise in the laboratory diagnosis of diseases that occur during fetal growth, infancy, and child development.
Education and Training
Tulane Univ Sch of Med, New Orleans La 1971
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Documentation of EWS gene rearrangements by fluorescence in-situ hybridization (FISH) in frozen sections of Ewing's sarcoma-peripheral primitive neuroectodermal tumor.
- Myc oncogene expression and nude mouse tumorigenicity and metastasis formation are higher in alveolar than embryonal rhabdomyosarcoma cell lines.
- MAT1-modulated CAK activity regulates cell cycle G(1) exit.
- DNA microarrays in pediatric cancer.
- Expression profiling of human tumors: the end of surgical pathology?
- PAX3-FKHR and PAX7-FKHR gene fusions are prognostic indicators in alveolar
- cDNA microarray analysis of global gene expression in sarcomas.
- Retinoid-induced G1 arrest and differentiation activation are associated with a switch to cyclin-dependent kinase-activating kinase hypophosphorylation of retinoic acid receptor alpha.
- Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters.
- Peripheral neuroepithelioma: a light microscopic, immunocytochemical, and ultrastructural study.
- Growth inhibition of breast epithelial cells by celecoxib is associated with upregulation of insulin-like growth factor binding protein-3 expression.
- MAT1-modulated cyclin-dependent kinase-activating kinase activity cross-regulates neuroblastoma cell G1 arrest and neurite outgrowth.
- Selective usage of D-Type cyclins by Ewing's tumors and rhabdomyosarcomas.
- Retinoid-modulated MAT1 ubiquitination and CAK activity.
- Lack of interferon response in animals to naked siRNAs.
Treatments
- Cancer, Sarcoma (bone And Soft Tissue Cancer)
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