Dr. Ronald Ray Morrison MD
Pediatrician | Pediatric Critical Care Medicine
332 N Lauderdale St Memphis TN, 38105About
Dr. Ronald Morrison is a pediatrician practicing in Memphis, TN. Dr. Morrison is a doctor who specializes in the health care of children. As a pedicatrician, Dr. Morrison diagnoses and treats infections, injuries, diseases and other disorders in children. Pediatricians typically work with infants, children, teenagers and young adults up to age 21. They practice medical care as well as preventative health care. Dr. Morrison can oversee and manage the physical, mental and emotional health of their patients.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Effect of cardiac A(1) adenosine receptor overexpression on sarcoplasmic reticulum function.
- A1 adenosine receptor overexpression decreases stunning from anoxia-reoxygenation: role of the mitochondrial K(ATP) channel.
- Genetic modulation of adenosine receptor function and adenosine handling in murine hearts: insights and issues.
- Adenosine receptors and the heart: role in regulation of coronary blood flow and cardiac electrophysiology.
- Impact of continuous renal replacement therapy on oxygenation in children with acute lung injury after allogeneic hematopoietic stem cell transplantation.
- Protection from doxorubicin-induced cardiomyopathy using the modified anthracycline N-benzyladriamycin-14-valerate (AD 198).
- Severe H1N1-associated acute respiratory failure in immunocompromised children.
- Impact of neutrophil recovery on oxygenation in pediatric oncology patients with acute hypoxemic respiratory failure.
- The adenosine A₂A receptor - myocardial protectant and coronary target in endotoxemia.
- Continuous renal replacement therapy in children post-hematopoietic stem cell transplantation: the present and the future.
- A Critical Care and Transplantation-Based Approach to Acute Respiratory Failure after Hematopoietic Stem Cell Transplantation in Children.
- Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium.
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