Dr. Wayne E Taylor MD
Pathologist | Anatomic Pathology & Clinical Pathology
7777 Forest Lane Dallas TX, 75230About
Dr. Wayne Taylor is a pathologist practicing in Dallas, TX. Dr. Taylor is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Taylor can help your primary care doctor make a diagnosis about your medical condition. Dr. Taylor may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Education and Training
University of Texas Southwestern Medical Center Southwestern Medical School 1973
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Androgens stimulate myogenic differentiation and inhibit adipogenesis in C3H 10T1/2 pluripotent cells through an androgen receptor-mediated pathway.
- Alteration of gene expression profiles in skeletal muscle of rats exposed to microgravity during a spaceflight.
- The mechanisms of androgen effects on body composition: mesenchymal pluripotent cell as the target of androgen action.
- Human KIT ligand promoter is positively regulated by HMGA1 in breast and ovarian cancer cells.
- Androgen receptor in human skeletal muscle and cultured muscle satellite cells: up-regulation by androgen treatment.
- Delta-4-androstene-3,17-dione binds androgen receptor, promotes myogenesis in vitro, and increases serum testosterone levels, fat-free mass, and muscle strength in hypogonadal men.
- Testosterone inhibits adipogenic differentiation in 3T3-L1 cells: nuclear translocation of androgen receptor complex with beta-catenin and T-cell factor 4 may bypass canonical Wnt signaling to down-regulate adipogenic transcription factors.
- The effect of glutamine on prevention of glucocorticoid-induced skeletal muscle atrophy is associated with myostatin suppression.
- Regulation of myogenic differentiation by androgens: cross talk between androgen receptor/ beta-catenin and follistatin/transforming growth factor-beta signaling pathways.
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