Dr. Maureen An-ping Su M. D.
Pediatrician
Ucsf Medical Center Childrens Hospital 505 Parnassuss Ave B San Francisco CA, 94143About
Dr. Maureen Su is a pediatrician practicing in San Francisco, CA. Dr. Su is a doctor who specializes in the health care of children. As a pedicatrician, Dr. Su diagnoses and treats infections, injuries, diseases and other disorders in children. Pediatricians typically work with infants, children, teenagers and young adults up to age 21. They practice medical care as well as preventative health care. Dr. Su can oversee and manage the physical, mental and emotional health of their patients.
Education and Training
Harvard Med Sch, Boston Ma 2000
Board Certification
PediatricsAmerican Board of PediatricsABP- Pediatric Endocrinology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- A screen for modifiers of decapentaplegic mutant phenotypes identifies lilliputian, the only member of the Fragile-X/Burkitt's Lymphoma family of transcription factors in Drosophila melanogaster.
- Aire: an update.
- Mechanisms of an autoimmunity syndrome in mice caused by a dominant mutation in Aire.
- Deletional tolerance mediated by extrathymic Aire-expressing cells.
- The role of X-linked FOXP3 in the autoimmune susceptibility of Turner Syndrome patients.
- Monogenic autoimmune diseases: insights into self-tolerance.
- A novel myelin P0-specific T cell receptor transgenic mouse develops a fulminant autoimmune peripheral neuropathy.
- Aire and T cell development.
- An essential role of the transcription factor GATA-3 for the function of regulatory T cells.
- Defective autoimmune regulator-dependent central tolerance to myelin protein zero is linked to autoimmune peripheral neuropathy.
- Aire deficiency promotes TRP-1-specific immune rejection of melanoma.
- Divergent effects of T cell costimulation and inflammatory cytokine production on autoimmune peripheral neuropathy provoked by Aire deficiency.
- Patients with antineutrophil cytoplasmic antibody-associated vasculitis have defective Treg cell function exacerbated by the presence of a suppression-resistant effector cell population.
- Reply: To PMID 23553415.
- Differential T cell responses to residual viral antigen prolong CD4+ T cell contraction following the resolution of infection.
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