Dr. Juan C Jaume MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
451 Junction Rd Madison WI, 53717About
Dr. Juan Jaume practices Endocrinology in Madison, WI. Dr. Jaume specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Jaume examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Universidad De Buenos Aires, Facultad De Ciencias Medicas MD
Provider Details
Expert Publications
Data provided by the National Library of Medicine- A mouse monoclonal antibody to a thyrotropin receptor ectodomain variant provides insight into the exquisite antigenic conformational requirement, epitopes and in vivo concentration of human autoantibodies.
- Cellular thyroid peroxidase (TPO), unlike purified TPO and adjuvant, induces antibodies in mice that resemble autoantibodies in human autoimmune thyroid disease.
- Rarity of autoantibodies to a major autoantigen, thyroid peroxidase, that interact with denatured antigen or with epitopes outside the immunodominant region.
- Lack of female bias in a mouse model of autoimmune hyperthyroidism (Graves' disease).
- Does GAD have a unique role in triggering IDDM?
- Development of overt autoimmune hyperthyroidism in a patient therapeutically immunosuppressed after liver transplantation.
- Cytokines, IgG subclasses and costimulation in a mouse model of thyroid autoimmunity induced by injection of fibroblasts co-expressing MHC class II and thyroid autoantigens.
- Isolation and characterization of human monoclonal autoantibodies to glutamic acid decarboxylase.
- Higher serum TSH in thyroid cancer patients occurs independent of age and correlates with extrathyroidal extension.
- T regulatory cells distinguish two types of primary hypophysitis.
- Homing of GAD65 specific autoimmunity and development of insulitis requires expression of both DQ8 and human GAD65 in transgenic mice.
- Glucose intolerance and diabetes following antigen-specific insulitis in diabetes-susceptible "humanized" transgenic mice.
- Hashimoto thyroiditis is more frequent than expected when diagnosed by cytology which uncovers a pre-clinical state.
- Symptomatic hyponatremia.
- Eukaryotic translation initiation factor 5A inhibition alters physiopathology and
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