Dr. Barbara E Murray M.D.
Infectious Disease Specialist | Infectious Disease
6410 Fannin St 604 Houston TX, 77030About
Dr. Barbara Murray is an infectious disease specialist practicing in Houston, TX. Dr. Murray specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Molecular typing of selected Enterococcus faecalis isolates: pilot study using multilocus sequence typing and pulsed-field gel electrophoresis.
- Involvement of PhoP-PhoS homologs in Enterococcus faecalis virulence.
- Evidence that the enterococcal polysaccharide antigen gene (epa) cluster is widespread in Enterococcus faecalis and influences resistance to phagocytic killing of E. faecalis.
- An Enterococcus faecalis ABC homologue (Lsa) is required for the resistance of
- Activity of tigecycline (GAR-936), a novel glycylcycline, against Enterococci in the mouse peritonitis model.
- A potential virulence gene, hylEfm, predominates in Enterococcus faecium of clinical origin.
- Clinical isolates of Enterococcus faecium exhibit strain-specific collagen binding mediated by Acm, a new member of the MSCRAMM family.
- Role of the Enterococcus faecalis GelE protease in determination of cellular chain length, supernatant pheromone levels, and degradation of fibrin and misfolded surface proteins.
- In vivo efficacy of the ketolide ABT-773 (cethromycin) against enterococci in a mouse peritonitis model.
- An Enterococcus faecium secreted antigen, SagA, exhibits broad-spectrum binding to extracellular matrix proteins and appears essential for E. faecium growth.
- Relapse of type A beta-lactamase-producing Staphylococcus aureus native valve endocarditis during cefazolin therapy: revisiting the issue.
- Translocation of Enterococcus faecalis strains across a monolayer of polarized human enterocyte-like T84 cells.
- Molecular epidemiology of the fsr locus and of gelatinase production among different subsets of Enterococcus faecalis isolates.
- Influence of origin of isolates, especially endocarditis isolates, and various genes on biofilm formation by Enterococcus faecalis.
- A family of putative MSCRAMMs from Enterococcus faecalis.
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