Steven M. Pogwizd M.D.
Cardiologist | Cardiovascular Disease
1670 University Blvd Vh B140 Birmingham AL, 35294About
Dr. Steven Pogwizd is a cardiologist practicing in Birmingham, AL. Dr. Pogwizd specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Pogwizd also practices preventative medicine, helping patients maintain a heart-healthy life.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Upregulation of Na(+)/Ca(2+) exchanger expression and function in an arrhythmogenic rabbit model of heart failure.
- Cycle length dynamics during ventricular tachycardia in the infarcted heart.
- Increased Na(+)-Ca(2+) exchanger in the failing heart.
- Arrhythmogenesis and contractile dysfunction in heart failure: Roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness.
- Calcium cycling in heart failure: the arrhythmia connection.
- Intracellular [Na+] and Na+ pump rate in rat and rabbit ventricular myocytes.
- Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing hearts.
- Intracellular Na(+) concentration is elevated in heart failure but Na/K pump
- Upregulated Na/Ca exchange is involved in both contractile dysfunction and arrhythmogenesis in heart failure.
- Na/Ca exchange in heart failure: contractile dysfunction and arrhythmogenesis.
- Clinical potential of sodium-calcium exchanger inhibitors as antiarrhythmic agents.
- Intracellular Na in animal models of hypertrophy and heart failure: contractile function and arrhythmogenesis.
- Prevention of ischemia-induced reentrant ventricular arrhythmias by a peptide that enhances gap junctional conductance.
- Elevated sarcoplasmic reticulum Ca2+ leak in intact ventricular myocytes from rabbits in heart failure.
- The contribution of nonreentrant mechanisms to malignant ventricular arrhythmias.
Fellowships
- Barnes-Jewish Hospital 1984
- Barnes-Jewish Hospital 1987
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