Dr. Colin James Henderson MD
Emergency Physician | Emergency Medical Services
19021 Hwy 285 Conejos County Hospi La Jara CO, 81140About
Dr. Colin Henderson practices Emergency Medicine in La Jara, CO. Dr. Henderson assesses patients who seek immediate medical attention at any time of day or night. Emergency Medicine Physicians are trained to efficiently work with each patient and situation no matter how acute or life-threatening. Dr. Henderson examines patients, determines means of testing, diagnoses conditions, and decides the best treatment methods.
Education and Training
Howard Univ Coll of Med, Washington Dc 1990
Board Certification
Family MedicineAmerican Board of Family MedicineABFM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Loss of the Nrf2 transcription factor causes a marked reduction in constitutive and inducible expression of the glutathione S-transferase Gsta1, Gsta2, Gstm1, Gstm2, Gstm3 and Gstm4 genes in the livers of male and female mice.
- Inactivation of the hepatic cytochrome P450 system by conditional deletion of hepatic cytochrome P450 reductase.
- Protein expression profiling of glutathione S-transferase pi null mice as a strategy to identify potential markers of resistance to paracetamol-induced toxicity in the liver.
- Increased constitutive c-Jun N-terminal kinase signaling in mice lacking glutathione S-transferase Pi.
- Identification of novel roles of the cytochrome p450 system in early embryogenesis: effects on vasculogenesis and retinoic Acid homeostasis.
- Knockout mice in xenobiotic metabolism.
- Transgenic analysis of human drug-metabolizing enzymes: preclinical drug development and toxicology.
- Transcription factor Nrf2 is essential for induction of NAD(P)H:quinone oxidoreductase 1, glutathione S-transferases, and glutamate cysteine ligase by broccoli seeds and isothiocyanates.
- Relationship between hepatic phenotype and changes in gene expression in cytochrome P450 reductase (POR) null mice.
- Environmental pollutant and potent mutagen 3-nitrobenzanthrone forms DNA adducts after reduction by NAD(P)H:quinone oxidoreductase and conjugation by acetyltransferases and sulfotransferases in human hepatic cytosols.
- Bioactivation of 3-aminobenzanthrone, a human metabolite of the environmental pollutant 3-nitrobenzanthrone: evidence for DNA adduct formation mediated by cytochrome P450 enzymes and peroxidases.
- Role of hepatic cytochrome p450s in the pharmacokinetics and toxicity of cyclophosphamide: studies with the hepatic cytochrome p450 reductase null mouse.
- The hepatic cytochrome P450 reductase null mouse as a tool to identify a successful candidate entity.
- Disruption of the glutathione transferase pi class genes.
- Hepatic cytochrome P-450 reductase-null mice show reduced transcriptional response to quercetin and reveal physiological homeostasis between jejunum and liver.
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