Dr. Ben Ho Park M.D.
Hematologist-Oncologist | Hematology & Oncology
3601 THE VANDERBILT CLINIC NASHVILLE TN, 37232About
Dr. Ben Park is a hematologist oncologist practicing in Baltimore, MD. Dr. Park specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Education and Training
Univ of Pa Sch of Med, Philadelphia Pa 1995
Perelman School of Medicine University of Pennsylvania 1996
Perelman School of Medicine at the University of Pennsylvania 1995
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- p21(WAF1/CIP1) mediates the growth response to TGF-beta in human epithelial cells.
- The PIK3CA gene is mutated with high frequency in human breast cancers.
- Duel nature of TGF-beta signaling: tumor suppressor vs. tumor promoter.
- Identification of compounds that inhibit growth of 2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine-resistant cancer cells.
- Physiologic estrogen receptor alpha signaling in non-tumorigenic human mammary epithelial cells.
- The consensus coding sequences of human breast and colorectal cancers.
- P21 gene knock down does not identify genetic effectors seen with gene knock out.
- Knock-in of mutant K-ras in nontumorigenic human epithelial cells as a new model for studying K-ras mediated transformation.
- The multiple myeloma associated MMSET gene contributes to cellular adhesion, clonogenic growth, and tumorigenicity.
- Tamoxifen-stimulated growth of breast cancer due to p21 loss.
- P21 and p27: roles in carcinogenesis and drug resistance.
- The PIK3CA gene as a mutated target for cancer therapy.
- Knockin of mutant PIK3CA activates multiple oncogenic pathways.
- Deletion of PTEN promotes tumorigenic signaling, resistance to anoikis, and altered response to chemotherapeutic agents in human mammary epithelial cells.
- PIK3CA and KRAS mutations predict for response to everolimus therapy: now that's RAD001.
Clinical Trials
Treatments
- Breast Cancer
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